age matched wild type (wt) controls Search Results


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Taconic Biosciences 129 sv/ev wild-type (wt) controls
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Jackson Laboratory b6.129f2 wild-type mice
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Taconic Biosciences nontransgenic colony control (wild-type, wt)
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Jackson Laboratory age-matched wt control mice
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Taconic Biosciences matched wild type (wt) controls (balb.b/ai-il6n9)
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Harlan Laboratories wild-type (wt) age-matched controls
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Jackson Laboratory colony-matched wild-type homozygous control mice
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Jackson Laboratory background-matched wild-type (wt) mice
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Jackson Laboratory wild-type (wt) control mice jackson #005304
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Jackson Laboratory age-matched male control (wt) mice
<t>Dystrophin</t> at the neuromuscular junction. Neuromuscular junctions (NMJs) were fluorescently stained with an acetylcholine receptor binding neurotoxin (α-Bungarotoxin, BTX, green), and antibodies against dystropin ( red ). Dystrophin, a stabilizing membrane protein, is present at the NMJ in healthy (wild-type, WT) tissue, but is missing in mdx mice. NMJs from mdx muscle also show drastic morphological differences when compared with NMJs from WT mice. It has been suggested that the lack of dystrophin is indirectly involved with the fragmented appearance of acetylcholine receptors seen in mdx mice. Scale bar equals 10 µm
Age Matched Male Control (Wt) Mice, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Dystrophin at the neuromuscular junction. Neuromuscular junctions (NMJs) were fluorescently stained with an acetylcholine receptor binding neurotoxin (α-Bungarotoxin, BTX, green), and antibodies against dystropin ( red ). Dystrophin, a stabilizing membrane protein, is present at the NMJ in healthy (wild-type, WT) tissue, but is missing in mdx mice. NMJs from mdx muscle also show drastic morphological differences when compared with NMJs from WT mice. It has been suggested that the lack of dystrophin is indirectly involved with the fragmented appearance of acetylcholine receptors seen in mdx mice. Scale bar equals 10 µm

Journal: Cellular and Molecular Life Sciences

Article Title: Recovery of altered neuromuscular junction morphology and muscle function in mdx mice after injury

doi: 10.1007/s00018-014-1663-7

Figure Lengend Snippet: Dystrophin at the neuromuscular junction. Neuromuscular junctions (NMJs) were fluorescently stained with an acetylcholine receptor binding neurotoxin (α-Bungarotoxin, BTX, green), and antibodies against dystropin ( red ). Dystrophin, a stabilizing membrane protein, is present at the NMJ in healthy (wild-type, WT) tissue, but is missing in mdx mice. NMJs from mdx muscle also show drastic morphological differences when compared with NMJs from WT mice. It has been suggested that the lack of dystrophin is indirectly involved with the fragmented appearance of acetylcholine receptors seen in mdx mice. Scale bar equals 10 µm

Article Snippet: We used age-matched male control (WT) and mdx (lacking dystrophin) mice from the C57BL/10ScSnJ strain (The Jackson Laboratory, Bar Harbor, ME).

Techniques: Staining, Binding Assay, Membrane